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Vol. 14. Issue 3.
Pages 97-99 (July - September 1978)
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Vol. 14. Issue 3.
Pages 97-99 (July - September 1978)
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Hemoptisis masiva en mineros del carbon
Massive hemoptysis in goal miners
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T. González Bazús*, J.A. Muñoz Martínez**, J.A. Mosquera Pestaña***
* Médico Residente. Instituto Nacional Je Silicosis. Oviedo
** Jefe Clínico de Neumología. Instituto Nacional Je Silicosis. Oviedo
*** Jefe del Servicio de Neumología. Instituto Nacional Je Silicosis. Oviedo
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Entre los 30225 mineros del carbón encontramos 12 pacientes con hemoptisis masiva (más de 600c.c./24 horas). Todos ellos varones de una edad de 52,81 años ± 12,04. 9 tenían neumoconiosis complicada y 3 no padecían neumoconiosis. El VEMS era de 1.721±811c.c. Los gases en sangre arterial en el momento de la hemoptisis etan: Pa O2 63.09mm de Hg. ± 8,85. Pa CO2 39,72mm. de Hg. ± 7,2 pH 7,41±0,058. 5 Pacienleslpresentaron shock.

La hemorragia pulmonar masiva ocurría en el 0,04% de la totalidad de los mineros estudiados. La etiología más frecuente fue la tuberculosis pulmonar que acontecía en 8 casos. (1,4% de los 564 tuberculosos bacteriológicamente probados). Las restantes causas: 1 micetoma, 1 estenosis mitral, 1 coagulopatia por insuficiencia hepatocelular grave y 1 neumonía necrotizante por bacteroides. La tuberculosis es, por tanto, la causa más frecuente de hemoptisis masiva (35 veces más frecuente en los tuberculosos que en los restantes mineros vistos en nuestro Centro.)

La decisión terapéutica fue quirúrgica en tres casos, dos de los cuales viven en la actualidad (mortalidad 33%). En los 9 pacientes restantes el tratamiento fue médico, sobreviviendo 2 de ellos (mortalidad 87, 5%).

A la vista de estos datos, deducimos que la etiología, tratamiento y mortalidad de la hemoptisis masiva en mineros del carbón son equiparables a los publicados en poblaciones no mineras.

Summary

Out ol a group of 30,225 coal miners the authors lound 12 patients with massive hemoptysis (more i han 600 cc./24 hours). All of them were male with an age of 52.81 years + 12.04. Nine suffered trom complex pnetiinoconiosis and three did not llave pnenmoconiosis. The VEMS were ol 1,721±811 ce. Gases in arterial blood at the time of hemoptysis were: PaO2 63.09 mmHg. + 8.85. PaCO2 39.72mmHg±7.2. pH 7.41±0.058. Five patients presented

shock.

Massive pulmonary hemorrhage occurred in 0.04% of the totality of the miners studied. The most freqnent etiology was pulmonary tuberculosis which happened in 8 cases. (1.4% of the 564 sujects with tuberculosis bacteriologically proved). The remaining causes were: 1 myeetoma, 1 mitral stenosis, 1 coagulopathv by grave hepatocellular insufficency and l necrotizing pneumemia by bacteroids. Tuberculosis is, therefore, the most frequent cause of massive hemoptysis (35 times more frequent for tubercular subjects than for tlte rest of the miners visited in the authors’ Center). The therapeutical decision vvas surgical in three cases, two of these patients eurrently 11 ve (mortality 33%). For the remaining 9 patients, the treatment was medical, tvvo of them survive (mortality 87,5%).

Taking these data into account, the authors deduce that the etiology, treatment and mortality of massive hemoptysis in coal miners are comparable lo those published for nonmining populations.

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Copyright © 1978. Sociedad Española de Neumología y Cirugía Torácica
Archivos de Bronconeumología
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